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Preston Ge
Preston Ge
MD/PhD student, Harvard Medical School
Verified email at hms.harvard.edu
Title
Cited by
Cited by
Year
Pathological α-synuclein transmission initiated by binding lymphocyte-activation gene 3
X Mao, MT Ou, SS Karuppagounder, TI Kam, X Yin, Y Xiong, P Ge, ...
Science 353 (6307), aah3374, 2016
6752016
PINK1 and Parkin mitochondrial quality control: a source of regional vulnerability in Parkinson’s disease
P Ge, VL Dawson, TM Dawson
Molecular neurodegeneration 15 (1), 20, 2020
3352020
Activation of tyrosine kinase c-Abl contributes to α-synuclein–induced neurodegeneration
S Brahmachari, P Ge, SH Lee, D Kim, SS Karuppagounder, M Kumar, ...
The Journal of clinical investigation 126 (8), 2970-2988, 2016
1642016
The cell biology of Parkinson’s disease
N Panicker, P Ge, VL Dawson, TM Dawson
Journal of Cell Biology 220 (4), e202012095, 2021
942021
c-Abl and Parkinson’s disease: mechanisms and therapeutic potential
S Brahmachari, SS Karuppagounder, P Ge, S Lee, VL Dawson, ...
Journal of Parkinson's Disease 7 (4), 589-601, 2017
822017
LRRK2 G2019S transgenic mice display increased susceptibility to 1-methyl-4-phenyl-1, 2, 3, 6-tetrahydropyridine (MPTP)-mediated neurotoxicity
SS Karuppagounder, Y Xiong, Y Lee, MC Lawless, D Kim, E Nordquist, ...
Journal of chemical neuroanatomy 76, 90-97, 2016
532016
Parkin interacting substrate zinc finger protein 746 is a pathological mediator in Parkinson’s disease
S Brahmachari, S Lee, S Kim, C Yuan, SS Karuppagounder, P Ge, R Shi, ...
Brain 142 (8), 2380-2401, 2019
492019
Aplp1 and the Aplp1-Lag3 Complex facilitates transmission of pathologic α-synuclein
X Mao, H Gu, D Kim, Y Kimura, N Wang, E Xu, H Wang, C Chen, S Zhang, ...
bioRxiv, 2021.05. 01.442157, 2021
112021
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